The Mikoshiba lab at ShanghaiTech University is recruiting ambitious and highly motivated individuals for two funded postdoctoral positions.
The Mikoshiba lab studies the cellular and molecular mechanism of calcium signalling in the normal and disease states. Successful candidates will participate in a stimulating, collaborative, and worldwide scientific environment with fully innovative equipment and facilities. The lab discovered a key calcium regulator, IP3 receptor (Nature 1989) and found its role in fertilization (Science 1992), dorso-ventral axis formation (Science 1997, Nature 2002), neurite extension (Science 2005), exocrine secretion (Science 2005), learning and memory (Nature 2000, Nature Neurosci 2015), behavior (Nature 1996), and apoptosis (eLIFE 2016). ?
Based on these findings, The Mikoshiba lab is aiming for cutting-edge research in science. Ideal candidates will have a PhD or equivalent and should have a strong background in neurobiology, cell biology, or molecular biology of calcium channel or signalling molecules. Expertise in developmental biology, calcium imaging, structural biology, electrophysiology, biochemistry, or biophysics would be welcome. One position will focus mainly on neurological or developmental functions of calcium signalling and the other position will focus on the biophysical mechanism of regulation or function of calcium signalling.
For more information, please click here.
Applicants should send CV as well as name and email of three references to Mikoshiba (mikosiba@shanghaitech.edu.cn)?and cc to: hr_siais@shanghaitech.edu.cn
Please refer to our already published papers:
Nature 299:357-59 (1982), Nature 342:32-8 (1989), Nature 350:398-402 (1991), Nature 353:566-69 (1991), Science 257:251-5 (1992), Cell 73:555-570 (1993), Nature 379:168-71 (1996), Science 278:1940-3 (1997), Nature 385:70-74 (1997), Nature 389:730-733 (1997), Science 279:237-242 (1998), Science 282:1705-1708 (1998), Science 287: 1647-51 (2000), Nature 408:584-588 (2000), Science 292:920-923 (2001), Nature Biotech 20:87-90 (2002), Nature 417:295-299 (2002), Cell 114:545-557 (2003), Cell 120:85-98 (2005), Science 309:2232-4 (2005), Molecular Cell 22:795-806 (2006), Molecular Cell 33:472-82 (2009), Nature Methods 7:729-732 (2010), Molecular Cell 58:1015-27 (2015), Nature Neurosci 18:698-707 (2015), Nature Commun (2016) DOI: 10.1038/ncomms11100
